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Even though the idea of limiting visitor group size has been generally accepted, the actual limit was not set based on definitive scientific evidence. Although no specific data relating disease exposure and group size has been found in the literature, it is possible to assess the weight of visitor group sizes on given gorilla groups, all other variables being equal. In other words, if we know that transmission is a direct function of exposure, the question is: how would a change in visitor group size affect gorilla exposure, ignoring other factors involved? Epidemiological facts reviewed above show that the degree of exposure is directly related to the number of infectious sources. This means that each additional visitor represents an additional potential source of infection, both direct and indirect via fomites, faeces, food, etc. ; . Before reviewing possible actions, two additional points need to be made. First, as noted earlier, the variables determining the risk of transmission from individual sources are not independent from each other. If all variables were independent, a target exposed to one single or 100 different sources each representing a probability of infection of say 1 in 10, would always have a 1 in chance of acquiring the infection from any one source, independent of the number of sources or the duration of exposure. However, practically or biologically speaking, a longer exposure increases the chances of contact between source and target, through fluctuations in either distance, wind, and target's gorillas ; or source's tourists ; behaviour. An increased number of sources also translates into a greater amount of potential virus or germ shedding, which directly increases the degree of exposure, hence the risk of transmission. The second point concerns the vulnerability of gorillas to human diseases, and the potential for spread of a disease once introduced into a group population. As mentioned in the preamble to this discussion, until further evidence is available on gorilla health and resistance to human diseases, one must approach the rules from a "catastrophe prevention" perspective. We must avoid the possibility of any single event that could greatly reduce or extinguish the remaining population. It must be assumed that gorillas are in a state of high vulnerability to human diseases, as shown in examples reviewed above in both wild and captive situations. Therefore the point is that if one individual gorilla is infected, the potential for spread in the group is related to the vulnerability - or the immune status - of the animals. An illustration of this is shown in Figure 1 and adalat. 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USA Genetics and genomics of Bipolar Disorder: Combining multiple data types and generating meaning from the mania Mary-Anne Enoch, K White, J Waheed, X-Z Hu, C Harris, D Goldman National Institute on Alcohol Abuse and Alcoholism, NIH, USA Differences in genetic origins and attentional responses between pure and comorbid anxiety disorders in humans Christopher Kliethermes, J Crabbe - University of California-San Francisco, USA Behavioral, pharmacological, and gene expression differences between lines of mice bred for differences in exploratory behavior David Segal, K Walder, M Malakellis, A Sanigorski, G Collier - Deakin University, Waurn Ponds, Australia Large scale gene expression analysis in the hypothalamus of an animal model of depression Douglas Swanson, E Brauer, G Ramirez, M Butler, E Polosetsky, Y Benjamini, I Golani, D Goldowitz - University of Tennessee, Memphis, USA A neuroethological approach to dissecting the roles of cerebellar purkinjie cells in open field behavior Michael Butterfield, C Rankin University of British Columbia, Canada - The effects of stimulation and ethanol exposure on long-term memory training in C. elegans 16: 00-18: 00 Sunday, May 21 07: 30-08: 00 SPEAKERS: Poster Session in Buchanan Lower Lobby and zyloprim.

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